Skip Nav Destination
1-6 of 6
Keywords: Cytochalasin BClose
J Exp Biol (2016) 219 (9): 1384–1393.
Published: 1 May 2016
... by those from short-horned sculpin at the same level of extracellular glucose. In Atlantic cod myocytes, glucose metabolism displayed what appears to be a saturable component with respect to extracellular glucose, and cytochalasin B inhibited glucose metabolism. These features revealed a facilitated...
J Exp Biol (2014) 217 (21): 3797–3804.
Published: 1 November 2014
... and glucose utilization determined by the two methodologies. As such, extracellular glucose is quite sufficient to fully satisfy the metabolic requirements of Atlantic cod RBCs. Under cytochalasin B inhibition of glucose uptake, the rates of glucose utilization and glycogenolysis fall short of matching...
J Exp Biol (2004) 207 (11): 1865–1874.
Published: 1 May 2004
... conditions. Working at basal levels, hearts did not require extracellular glucose to maintain power under aerobic conditions. However, cardiac performance was significantly reduced without exogenous glucose under oxygen-limiting conditions. The addition of the glucose transporter inhibitor cytochalasin B...
J Exp Biol (2002) 205 (14): 2089–2097.
Published: 15 July 2002
... phagocytosis than from mechanical stimulation of the pouch. Cytochalasin B (10 μg ml -1 ) inhibits phagocytosis in both microstomes and macrostomes, indicating that actin filaments play an active role in phagocytosis in both cell types. The antitubulin drug nocodazole(0.3-30 μmol l -1 ) inhibits the formation...
J Exp Biol (1997) 200 (22): 2871–2880.
Published: 1 November 1997
...-deoxyglucose (2-DG) by ventricle strips at 15 °C was increased by 45 % by anoxia and by 85 % by contractile activity over basal conditions. The anoxia- and contraction-induced increase in basal 2-DG uptake was inhibited completely by 25 μmol l −1 cytochalasin B, suggesting that facilitated glucose transporters...
J Exp Biol (1995) 198 (2): 577–583.
Published: 1 February 1995
... to 3-OMG. Although transport is inhibited by cytochalasin B and phloretin, the lack of saturation kinetics for transport in a physiologically relevant concentration range suggests either that a glucose transporter does not exist or that its affinity for glucose is extremely low. The marked increase...