There were errors published in J. Cell Sci. (2017) 130, 3818-3828 (doi: 10.1242/jcs.203646).

The incorrect actin loading controls were used for Fig. 1A and Fig. 3C. The corrected figure panels are shown below.

The authors apologise to readers for any inconvenience caused.

Fig. 1.
Fig. 1. Overexpression of LRRC8B attenuates the ATP-induced [Ca2+]c rise. (A) Western blot showing the overexpression of LRRC8B in HEK293 cells following transient transfection. β-actin was used as a loading control.
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Overexpression of LRRC8B attenuates the ATP-induced [Ca2+]c rise. (A) Western blot showing the overexpression of LRRC8B in HEK293 cells following transient transfection. β-actin was used as a loading control.

Fig. 1.
Fig. 1. Overexpression of LRRC8B attenuates the ATP-induced [Ca2+]c rise. (A) Western blot showing the overexpression of LRRC8B in HEK293 cells following transient transfection. β-actin was used as a loading control.
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Overexpression of LRRC8B attenuates the ATP-induced [Ca2+]c rise. (A) Western blot showing the overexpression of LRRC8B in HEK293 cells following transient transfection. β-actin was used as a loading control.

Fig. 3.
Fig. 3. LRRC8B regulates the ER Ca2+ pool. (C) Western blot showing the siRNA (50 nM)-mediated knockdown of endogenous LRRC8B expression in HEK293 cells. β-actin was used as an internal control. The expression level of LRRC8A was not affected.
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LRRC8B regulates the ER Ca2+ pool. (C) Western blot showing the siRNA (50 nM)-mediated knockdown of endogenous LRRC8B expression in HEK293 cells. β-actin was used as an internal control. The expression level of LRRC8A was not affected.

Fig. 3.
Fig. 3. LRRC8B regulates the ER Ca2+ pool. (C) Western blot showing the siRNA (50 nM)-mediated knockdown of endogenous LRRC8B expression in HEK293 cells. β-actin was used as an internal control. The expression level of LRRC8A was not affected.
View largeDownload slide

LRRC8B regulates the ER Ca2+ pool. (C) Western blot showing the siRNA (50 nM)-mediated knockdown of endogenous LRRC8B expression in HEK293 cells. β-actin was used as an internal control. The expression level of LRRC8A was not affected.

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© 2018. Published by The Company of Biologists Ltd
2018