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Dale W. Laird
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Journal Articles

Mutant Cx30-A88V mice exhibit hydrocephaly and sex-dependent behavioral abnormalities, implicating a functional role for Cx30 in the brain

In collection:
Developmental Disorders
Nicole M. Novielli-Kuntz, Eric R. Press, Kevin Barr, Marco A. M. Prado, Dale W. Laird
Journal: Disease Models & Mechanisms
Dis Model Mech (2021) 14 (1): dmm046235.
DOI: https://doi.org/10.1242/dmm.046235
Published: 26 January 2021
View article titled, Mutant Cx30-A88V mice exhibit hydrocephaly and sex-dependent behavioral abnormalities, implicating a functional role for Cx30 in the brain
Click here to open pdf in another window PDF for Mutant Cx30-A88V mice exhibit hydrocephaly and sex-dependent behavioral abnormalities, implicating a functional role for Cx30 in the brain
Journal Articles

An atrial-fibrillation-linked connexin40 mutant is retained in the endoplasmic reticulum and impairs the function of atrial gap-junction channels

Yiguo Sun, Xiaoling Tong, Honghong Chen, Tao Huang, Qing Shao, Weixiong Huang, Dale W. Laird, Donglin Bai
Journal: Disease Models & Mechanisms
Dis Model Mech (2014) 7 (5): 561–569.
DOI: https://doi.org/10.1242/dmm.013813
Published: 1 May 2014
View article titled, An atrial-fibrillation-linked connexin40 mutant is retained in the endoplasmic reticulum and impairs the function of atrial gap-junction channels
Click here to open pdf in another window PDF for An atrial-fibrillation-linked connexin40 mutant is retained in the endoplasmic reticulum and impairs the function of atrial gap-junction channels
Journal Articles

Oogenesis defects in a mutant mouse model of oculodentodigital dysplasia

Dan Tong, Deanne Colley, Renee Thoo, Tony Y. Li, Isabelle Plante, Dale W. Laird, Donglin Bai, Gerald M. Kidder
Journal: Disease Models & Mechanisms
Dis Model Mech (2009) 2 (3-4): 157–167.
DOI: https://doi.org/10.1242/dmm.000935
Published: 25 February 2009
View article titled, Oogenesis defects in a mutant mouse model of oculodentodigital dysplasia
Click here to open pdf in another window PDF for Oogenesis defects in a mutant mouse model of oculodentodigital dysplasia