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Cover image
Cover Image
Cover: Histological section of YBR/EiJ mouse eye displaying hallmarks of glaucoma, including optic nerve excavation and thinning of the retinal nerve fiber layer. The article by Nair et al. on page 863 presents a detailed characterization of YBR/EiJ inbred mice as a new inherited model of glaucoma. In YBR mice, two genetic loci act through distinct pathogenic processes in an additive manner to elevate intraocular pressure (IOP). Elevated IOP is a causal risk factor for glaucoma. Importantly, the locus on YBR chromosome 17 elevates IOP without any major blockage of the ocular drainage tissue. In this way, the effects of the chromosome 17 locus resemble open-angle glaucoma, a common form of glaucoma. Image provided by K. S. Nair and licenced under a Creative Commons Attribution 4.0 International licence.
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Call for papers – In Vitro Models of Human Disease to Inform Mechanism and Drug Discovery
We invite you to submit your latest research to our upcoming special issue: In Vitro Models of Human Disease to Inform Mechanism and Drug Discovery, coordinated by DMM Editor Vivian Li (The Francis Crick Institute, London, UK), alongside Guest Editors Austin Smith (University of Exeter, UK) and Joseph Wu (Stanford University School of Medicine, USA).The deadline for submitting articles is 6 October 2025.
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