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NARROW
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1-15 of 15
Keywords: KIT
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Journal Articles
In collection:
Reproductive biology
Chiara Capponi, Martina Palazzoli, Sara Di Persio, Stefania Fera, Gustavo Spadetta, Giorgio Franco, Joachim Wistuba, Stefan Schlatt, Nina Neuhaus, Dirk de Rooij, Elena Vicini
Journal:
Development
Development (2023) 150 (10): dev201430.
Published: 24 May 2023
.... , De Rooij , D. G. and Van Pelt , A. M. ( 1999 ). Differential expression of c-kit in mouse undifferentiated and differentiating type A spermatogonia . Endocrinology 140 , 5894 - 5900 . 10.1210/endo.140.12.7172 Shami , A. N. , Zheng , X. , Munyoki , S. K. , Ma , Q...
Includes: Supplementary data
Journal Articles
In collection:
Stem cells & regeneration
Alison J. May, Noel Cruz-Pacheco, Elaine Emmerson, Eliza A. Gaylord, Kerstin Seidel, Sara Nathan, Marcus O. Muench, Ophir D. Klein, Sarah M. Knox
Journal:
Development
Development (2018) 145 (21): dev166363.
Published: 5 November 2018
... that two ductal progenitor populations, marked exclusively by KRT14 and KIT, maintain non-overlapping ductal compartments after radiation exposure but do so through distinct cellular mechanisms. KRT14 + progenitor cells are fast-cycling cells that proliferate in response to radiation-induced damage...
Includes: Supplementary data
Journal Articles
Sara Di Persio, Rossana Saracino, Stefania Fera, Barbara Muciaccia, Valentina Esposito, Carla Boitani, Bartolomeo P. Berloco, Francesco Nudo, Gustavo Spadetta, Mario Stefanini, Dirk G. de Rooij, Elena Vicini
Journal:
Development
Development (2017) 144 (19): 3430–3439.
Published: 1 October 2017
... of spermatogonia. MAGE-A4 marks all spermatogonia, KIT marks all B spermatogonia and UCLH1 all Apale-dark (Ap-d) spermatogonia. We suggest that at the start of the spermatogenic lineage there are Ap-d spermatogonia that are GFRA1 High , likely including the spermatogonial stem cells. Next, UTF1 becomes expressed...
Includes: Supplementary data
Journal Articles
In collection:
Stem cells & regeneration
Bradley J. Goldstein, Garrett M. Goss, Rhea Choi, Dieter Saur, Barbara Seidler, Joshua M. Hare, Nirupa Chaudhari
Journal:
Development
Development (2016) 143 (23): 4394–4404.
Published: 1 December 2016
... for expression of c-KIT. We show that c-KIT signaling regulates self-renewal capacity and prevents neurodifferentiation in culture. Inhibition of TGFβ family signaling, a known negative regulator of embryonic basal cells, is also necessary for maintenance of the proliferative, undifferentiated state in vitro...
Includes: Supplementary data
Journal Articles
Journal:
Development
Development (2016) 143 (13): 2311–2324.
Published: 1 July 2016
... morphogenesis and differentiation into proacini by suppressing Kit expression through the upregulation of the transcription factor MYB, and concomitantly increasing the expression of distal progenitor markers. In addition, WNT signaling at the early stage of SMG development promoted end bud cell proliferation...
Includes: Supplementary data
Journal Articles
Journal:
Development
Development (2013) 140 (5): 996–1002.
Published: 1 March 2013
... be divided into establishment of the MSC, recruitment of the MSC to produce committed daughter cells, and the proliferation, differentiation and survival of these daughter cells. Reduction of Kit signaling results in dose-dependent reduction of melanocytes during larval regeneration. Here, we use clonal...
Includes: Supplementary data
Journal Articles
Journal:
Development
Development (2013) 140 (2): 280–290.
Published: 15 January 2013
... by an undifferentiated spermatogonial population and transition to a differentiating state involves attained expression of the KIT receptor. We found that impaired function of the X chromosome-clustered microRNAs 221 and 222 (miR-221/222) in mouse undifferentiated spermatogonia induces transition from a KIT – to a KIT...
Includes: Supplementary data
Journal Articles
Journal:
Development
Development (2007) 134 (6): 1081–1090.
Published: 15 March 2007
... metamorphic (LM) melanophores arise already within stripes. EM melanophores require the kit receptor tyrosine kinase, as kit mutants lack these cells but retain LM melanophores, which form a residual stripe pattern. To see if similar cell populations and genetic requirements are present in other species, we...
Includes: Multimedia, Supplementary data
Journal Articles
Shosei Yoshida, Mamiko Sukeno, Toshinori Nakagawa, Kazuyuki Ohbo, Go Nagamatsu, Toshio Suda, Yo-ichi Nabeshima
Journal:
Development
Development (2006) 133 (8): 1495–1505.
Published: 15 April 2006
... heparin. The detailed protocol is available upon request. The antibody detection and colorization was carried out as on sections, using an AP-conjugated anti-DIG antibody and BM purple AP substrate (Roche). Testicular sections of young mice were double stained for Kit or Ngn3 and galectin 1...
Includes: Supplementary data
Journal Articles
Journal:
Development
Development (2001) 128 (11): 1943–1949.
Published: 1 June 2001
...John F. Rawls; Stephen L. Johnson Embryonic neural crest-derived melanocytes and their precursors express the kit receptor tyrosine kinase and require its function for their migration and survival. However, mutations in kit also cause deficits in melanocytes that make up adult pigment patterns...
Journal Articles
Journal:
Development
Development (2000) 127 (17): 3715–3724.
Published: 1 September 2000
... in the regenerating fin and their requirement for the kit receptor tyrosine kinase. Using pre-existing melanin as a lineage tracer, we show that most fin regeneration melanocytes develop from undifferentiated precursors, rather than from differentiated melanocytes. Mutational analysis reveals two distinct classes...
Journal Articles
Takahiro Kunisada, Hisahiro Yoshida, Hidetoshi Yamazaki, Akitomo Miyamoto, Hiroaki Hemmi, Emi Nishimura, Leonard D. Shultz, Shin-Ichi Nishikawa, Shin-Ichi Hayashi
Journal:
Development
Development (1998) 125 (15): 2915–2923.
Published: 1 August 1998
...Takahiro Kunisada; Hisahiro Yoshida; Hidetoshi Yamazaki; Akitomo Miyamoto; Hiroaki Hemmi; Emi Nishimura; Leonard D. Shultz; Shin-Ichi Nishikawa; Shin-Ichi Hayashi ABSTRACT Mutations at the murine dominant white spotting ( Kit W ) and steel (Mgf Sl ) loci, encoding c-Kit receptor kinase and its...
Journal Articles
Karin Opdecamp, Atsuo Nakayama, Minh-Thanh T. Nguyen, Colin A. Hodgkinson, William J. Pavan, Heinz Arnheiter
Journal:
Development
Development (1997) 124 (12): 2377–2386.
Published: 15 June 1997
... with some alleles to complete absence of mature melanocytes with others. In the trunk region of wild-type embryos, Mitf- expressing cells that coexpressed the melanoblast marker Dct and the tyrosine kinase receptor Kit were found in the dorsolateral neural crest migration pathway. In contrast, in embryos...
Journal Articles
Journal:
Development
Development (1997) 124 (1): 65–77.
Published: 1 January 1997
... alter the coding sequence of the Kit receptor tyrosine kinase, resulting in a receptor with impaired kinase activity, or affect Kit expression. Here we describe the molecular and cell-type-specific developmental defects of two of the latter class of regulatory W alleles, W 57 and W banded(bd) . In both...
Journal Articles
Journal:
Development
Development (1996) 122 (11): 3597–3605.
Published: 1 November 1996
... transient (B6-Y AKR ) or severe (B6-Y TIR ) XY DOM sex reversal. We report that a point mutation, the dominant white spotting oncogene allele, Kit W-42J , exacerbates XY DOM sex reversal. In B6-Y AKR , penetrance of the trait is low; however, in B6-Y TIR , Kit W-42J exacerbated sex reversal...