There was an error in Development (2024) 151, dev202579 (doi:10.1242/dev.202579).

There were several errors in the supplementary file for this article.

The legend to Fig. S1A contained errors. The corrected and original legends are shown below.

Fig. S1 (corrected). Dis3 is required for maintenance of early spermatogenic lineage. (A) Exon map of the mouse Dis3 locus based on Ensembl (ENSMUSG00000033166). Two guide RNAs (gRNAs) were designed and two loxP sites were inserted as shown in the map. Exons 4 to 6 were deleted after crossing with a Cre transgenic mouse line leading to a frame shift mutation of Dis3 gene.

Fig. S1 (original). Dis3 is required for maintenance of early spermatogenic lineage. (A) Exon map of the mouse Dis3 locus based on Ensembl (ENSMUSG00000033166). Two guide RNAs (gRNAs) were designed and two loxP sites were inserted as shown in the map. Exons 4 to 6 were deleted after crossing with a Ddx4-cre transgenic mouse line, leading to a frame shift mutation of Dis3 gene.

Figure S3 and its legend contained errors. The corrected and original figures and legends are shown below.

Fig. S3 (corrected).

RNA-seq analysis of P4 control and Dis3 cKO testes. (A) GO enrichment analysis of up-regulated transcripts in P4 Dis3 cKO testes. GO terms were obtained by DAVID analysis. P value was obtained from the data by DAVID analysis. (B) KEGG pathway analysis of up-regulated genes in Dis3 mutant testes. The enriched terms were obtained by DAVID analysis. P value was obtained from the data by DAVID analysis. (C) Same as (A), but for down-regulated transcripts. (D) Same as (B), but for down-regulated genes. (E) An overview of the gene biotypes for these differentially expressed PROMPTs-associated transcripts shown in Figure 6E.

Fig. S3 (corrected).

RNA-seq analysis of P4 control and Dis3 cKO testes. (A) GO enrichment analysis of up-regulated transcripts in P4 Dis3 cKO testes. GO terms were obtained by DAVID analysis. P value was obtained from the data by DAVID analysis. (B) KEGG pathway analysis of up-regulated genes in Dis3 mutant testes. The enriched terms were obtained by DAVID analysis. P value was obtained from the data by DAVID analysis. (C) Same as (A), but for down-regulated transcripts. (D) Same as (B), but for down-regulated genes. (E) An overview of the gene biotypes for these differentially expressed PROMPTs-associated transcripts shown in Figure 6E.

Fig. S3 (original).

RNA-seq analysis of P4 control and Dis3 cKO testes. (A) GO enrichment analysis of up-regulated transcripts in P4 Dis3 cKO testes. GO terms were obtained by DAVID analysis. P value was obtained from the data by DAVID analysis. (B) KEGG pathway analysis of up-regulated genes in Dis3 mutant testes. The enriched terms were obtained by DAVID analysis. P value was obtained from the data by DAVID analysis. (C) Same as (A), but for down-regulated transcripts. (D) Same as (B), but for down-regulated genes. (E) An overview of the gene biotypes for these differentially expressed PROMPTs-associated transcripts shown in Figure 5E.

Fig. S3 (original).

RNA-seq analysis of P4 control and Dis3 cKO testes. (A) GO enrichment analysis of up-regulated transcripts in P4 Dis3 cKO testes. GO terms were obtained by DAVID analysis. P value was obtained from the data by DAVID analysis. (B) KEGG pathway analysis of up-regulated genes in Dis3 mutant testes. The enriched terms were obtained by DAVID analysis. P value was obtained from the data by DAVID analysis. (C) Same as (A), but for down-regulated transcripts. (D) Same as (B), but for down-regulated genes. (E) An overview of the gene biotypes for these differentially expressed PROMPTs-associated transcripts shown in Figure 5E.

Figure S6 contained errors. The corrected and original figures are shown below.

Fig. S6 (corrected).

GO and pathway enrichment analyses in different spermatogonial subtypes. (A) GO enrichment analysis of biological process categories of Dis3 cKO spermatogonia compared with control spermatogonia in SPG1, SPG2, SPG3 and SPG4 subtype by using Gene Set Enrichment Analysis (GSEA) software and Molecular Signatures Database (MSigDB) C5 GO gene sets. (B) Pathway enrichment analysis of Dis3 cKO spermatogonia compared with control spermatogonia in SPG1, SPG2, SPG3 and SPG4 cluster by using GSEA software and the MSigDB C2 curated gene sets.

Fig. S6 (corrected).

GO and pathway enrichment analyses in different spermatogonial subtypes. (A) GO enrichment analysis of biological process categories of Dis3 cKO spermatogonia compared with control spermatogonia in SPG1, SPG2, SPG3 and SPG4 subtype by using Gene Set Enrichment Analysis (GSEA) software and Molecular Signatures Database (MSigDB) C5 GO gene sets. (B) Pathway enrichment analysis of Dis3 cKO spermatogonia compared with control spermatogonia in SPG1, SPG2, SPG3 and SPG4 cluster by using GSEA software and the MSigDB C2 curated gene sets.

Fig. S6 (original).

GO and pathway enrichment analyses in different spermatogonial subtypes. (A) GO enrichment analysis of biological process categories of Dis3 cKO spermatogonia compared with control spermatogonia in SPG1, SPG2, SPG3 and SPG4 subtype by using Gene Set Enrichment Analysis (GSEA) software and Molecular Signatures Database (MSigDB) C5 GO gene sets. (B) Pathway enrichment analysis of Dis3 cKO spermatogonia compared with control spermatogonia in SPG1, SPG2, SPG3 and SPG4 cluster by using GSEA software and the MSigDB C2 curated gene sets.

Fig. S6 (original).

GO and pathway enrichment analyses in different spermatogonial subtypes. (A) GO enrichment analysis of biological process categories of Dis3 cKO spermatogonia compared with control spermatogonia in SPG1, SPG2, SPG3 and SPG4 subtype by using Gene Set Enrichment Analysis (GSEA) software and Molecular Signatures Database (MSigDB) C5 GO gene sets. (B) Pathway enrichment analysis of Dis3 cKO spermatogonia compared with control spermatogonia in SPG1, SPG2, SPG3 and SPG4 cluster by using GSEA software and the MSigDB C2 curated gene sets.

The online PDF of the supplementary file has been corrected.

The authors apologise for these errors, which do not impact the results and conclusions of the paper.