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Keywords: Host-pathogen interaction
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Journal Articles

BBQ methods: streamlined workflows for bacterial burden quantification in infected cells by confocal microscopy

Jacques Augenstreich, Michael Shuster, Yongqiang Fan, Zhihui Lyu, Jiqiang Ling, Volker Briken
Journal: Biology Open
Biol Open (2024) 13 (1): bio060189.
https://doi.org/10.1242/bio.060189
Published: 22 January 2024
...Jacques Augenstreich; Michael Shuster; Yongqiang Fan; Zhihui Lyu; Jiqiang Ling; Volker Briken ABSTRACT Accurate quantification of bacterial burden within macrophages, termed bacterial burden quantification (BBQ), is crucial for understanding host–pathogen interactions. Various methods have been...
View articletitled, BBQ methods: streamlined workflows for bacterial burden quantification in infected cells by confocal microscopy
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Includes: Supplementary data
Journal Articles

From pathogen to a commensal: modification of the Microbacterium nematophilum–Caenorhabditis elegans interaction during chronic infection by the absence of host insulin signalling

Maria Gravato-Nobre, Jonathan Hodgkin, Petros Ligoxygakis
Journal: Biology Open
Biol Open (2020) 9 (10): bio053504.
https://doi.org/10.1242/bio.053504
Published: 7 October 2020
... of C . elegans has permitted a glimpse of the defence mechanisms employed by the worm as well as the host–pathogen interactions triggering such mechanisms (see Hodgkin et al., 2000 , 2013 ; Nicholas and Hodgkin, 2004 ). One such pathogen is Microbacterium nematophilum ( Hodgkin et al., 2000...
View articletitled, From pathogen to a commensal: modification of the Microbacterium nematophilum–Caenorhabditis elegans interaction during chronic infection by the absence of host insulin signalling
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Includes: Supplementary data
Journal Articles

Mycobacterium tuberculosis Cpn60.2 (GroEL2) blocks macrophage apoptosis via interaction with mitochondrial mortalin

Sunil Joseph, Alex Yuen, Vijender Singh, Zakaria Hmama
Journal: Biology Open
Biol Open (2017) 6 (4): 481–488.
https://doi.org/10.1242/bio.023119
Published: 13 March 2017
... mycobacterial surface and traffics towards mitochondria. Once there, Cpn60.2 interacts with host mortalin leading to inhibition of macrophage's apoptosis. Host-pathogen interaction Phagosome Intra-cellular trafficking Mycobacterial persistence A key feature of tuberculosis (TB) pathogenesis...
View articletitled, Mycobacterium tuberculosis Cpn60.2 (GroEL2) blocks macrophage apoptosis via interaction with mitochondrial mortalin
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Includes: Supplementary data