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Cover image
Cover Image
Human mutations affecting the RPGRIP1 interactome cause severe ocular-renal diseases. Disease mutations in RPGRORF15 (red) and its partner, RPGRIP1α1 (green), promote their subcellular uncoupling and delocalizationthroughout the cytosolic compartment (background image). Foreground image shows an axial view along thecentral shaft of the propeller structure of the RCC1-homogous domain (RHD) of RPGR isoforms (red ribbon)superimposed on the template and known structure of RCC1 (yellow ribbon). See the article by Patil et al. (doi: 10.1242/bio.2011489).
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Fast & Fair peer review
We have recently launched the next phase of our Fast & Fair peer review initiative: offering high-quality peer review within 7 working days. To learn more about our progress and future plans, read the Editorial by our Editor-in-Chief Daniel Gorelick or visit the Fast & Fair peer review page.
The Company of Biologists Workshops
For the last 15 years, our publisher, The Company of Biologists, has provided an apt environment to inspire biology and support biologists through our Workshops series. Read about the evolution of the Workshop series and revisit JEB's experience with hosting the first Global South Workshop.
Daniel Gorelick on Open Access and Read & Publish in Biology Open
The remarkable cellular diversity of the African turquoise killifish telencephalon
African turquoise killifish have a remarkably short natural lifespan and their central nervous system has human-like ageing. Caroline Zandecki and colleagues reveal the cellular diversity during the explosive growth of the telencephalon.
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